World Aids Day - Time To Rethink Prevention, Treatment?
1 December is World Aids Day - time to think about Aids and the virus that is thought by orthodox medicine to be selectively devastating the immune system of ... mainly gays, intravenous drug users and the poor both in the West and in developing countries. Aids campaigners say we need to "raise awareness" about what they describe as a sexually transmitted disease. They say we need to invest more money to bring drugs to those in the developing countries who can't afford them.
Others are convinced that we should be helping to improve living conditions - sanitation, clean drinking water and basic nutrition. They add that treating the single diseases such as malaria and dysentery and a host of other infections prevalent in poor Africa and rural India would go a long way in eliminating "Aids".
Aids Rethinkers or Aids Denialists?Those suggesting to abandon the fixation on a virus never properly isolated and on highly toxic medications that cause many of the symptoms of "Aids" call themselves Aids rethinkers. A less complimentary term, used by the pro-Aids campaigners to position the independent thinkers is "Aids-denialists". Which of the two camps has truth on their side is hard to say for a casual observer. Greg Hambrick of the Charleston City Paper in a recent article titled Rethinking AIDS - Doubters abandon traditional HIV/AIDS theories and treatment strikes a balance between the two sides and gives an understandable introduction to their respective arguments.
Illustration - Charleston City Paper
Liam Scheff, an independent journalist who exposed scandalous experiments with toxic Aids drugs in a New York home for orphans and wards of the State, has been called an Aids denialist. His articles Inside Incarnation and The Truth about Nevirapine are indictments against human indifference and pharmaceutical profiteering at the cost of lives and untold suffering.Scheff's investigative work has triggered the ire of professional defenders of Aids orthodoxy, of those who sustain the viral paradigm and the chemical solution to the problem. In Correcting The AIDS Lies - Who Is Nick Bennett? Liam challenges one of these self-styled experts who pounce on what they perceive as the enemies of the infective-Aids paradigm.
Is Scheff an Aids denialist because he investigated and published his findings? Andrew Maniotis, Ph.D. a program director at the University of Illinois Department of Pathology, Anatomy and Cell Biology does not think so. In his paper The ABC's of AIDS denialism (it's a rather largish PDF file) Maniotis points up a myriad of inconsistencies in the official dogmatic stance which, summed up, could be described as HIV = AIDS = Retroviral Drugs = Delayed Death. Maniotis takes Montagnier, Gallo and a great number of other supporters of Aids orthodoxy to task for what he says are obvious contradictions and flawed arguments in the science of Aids. It's them who are the denialists, argues Maniotis in a very tongue-in-cheek paper.
In a more succinct but not less convincing way, Malaysian researcher Beldeu Singh sums up the facts of Aids-as-an-infection vs. Aids seen as a condition of cellular stress. His article, Don't Question the Gallo-HIV AIDS Dogma, is in the second part of this post. Beldeu has also written a celebratory piece - showing how Gallo himself has demonstrated that HIV does not attack T-cells and therefore cannot be the cause of Aids:
One trait the Aids rethinkers cum "denialists" have in common is a plea for real prevention and for standard medical treatment of the specific, known diseases that set in when the immune defenses have been down for a considerable period of time.
Real preventionOfficially, Aids prevention involves sexual abstinence, condoms, and even - who would have thought it - circumcision. The use of a cocktail of toxic drugs is also advocated, but there is no word of improving the economic outlook in developing nations, of bettering the living conditions and providing basic nutrition to increase the population's natural resistance. No word of nutrients like selenium and the three amino acids that are found to be low in most Aids patients. Geo-epidemiologist Harold Foster has identified these specific deficiencies as the likely culprit for increased susceptibility to contracting immune deficiency.
Harold Foster - What Really Causes AIDS
But those things are considered inappropriate to even seriously research. No wonder - they are not expected to bring great profits. As a matter of fact, they would tend to ruin a perfectly good business.But the ranks of rethinkers and "denialists" are steadily swelling. Quietly, they are making progress, even without funding from Bill and Melinda. Small scale trials and not-so-small-scale pilot programs to supply needed nutrients such as the efforts of the Dr Rath Foundation in South Africa are showing positive results, derisive hoots and cries by the real denialists notwithstanding.
Don't Question the Gallo-HIV AIDS Dogma, says Beldeu Singh from Malaysia, as he proceeds to tell us how this dogma is full of holes ...... and at the end of this post, you will also find a World Aids Day Message from Dr Leo Rebello.
With all these inconsistencies coming to the surface, this may indeed be a time for re-thinking Aids and our relation to health and disease.
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Image credit - Emma Holister
DON’T QUESTION THE GALLO-HIV AIDS DOGMA
Beldeu SinghAccording to the HIV theory of AIDS, the virus is sexually transmitted and that should produce an HIV-AIDS explosion in the heterosexual population within 25 years. But it did not happen.
This point is well put forth by Dr. Robert Root-Bernstein. Female prostitutes often have 200-300 sexual partners per year and are therefore assumed to have much higher rates of exposure to HIV and AIDS than the vast majority of heterosexuals. Many AIDS researchers assumed that female prostitutes would be the vectors (or means of transmission) of HIV and AIDS to the heterosexual community based on the fact that a single HIV-infected intravenous drug user or bisexual man could infect one female prostitute, who in turn could infect dozens or perhaps even hundreds of non-drug using heterosexual men. These men could, in turn, infect their other sexual partners, and an explosion of HIV and AIDS could occur among people without any obvious risk for AIDS. Paradoxically, no heterosexual epidemic has occurred and no evidence of female prostitutes transmitting HIV or AIDS into the heterosexual community exists for any Western nation. Transmission almost always seems to be drug related. In fact, sexual acquisition of HIV and AIDS among female prostitutes themselves is almost unknown in the absence of concomitant intravenous drug use. Cell-free viral particles have never been found directly in semen. In ‘American Journal of Epidemiology’ (Vol. 146, No.4), Nancy S. Padian et al reported:
“We estimate that HIV infectivity for male-to-female transmission is low, approximately 0.0009 per contact, and that infectivity for female-to-male transmission is even lower.”In New York City, for example, 40 to 50 percent of streetwalkers (a very low caste of prostitute) who have used IV drugs over the past decade are HIV seropositive. Among call girls in New York City (a higher caste of prostitute), no seropositivity was found among those who were drug free. These figures were constant between 1984 and 1989. These statistics have significant implications on the causative factor or factors of AIDS.
The biggest problem in the gallo-HIV theory which says that a pathogenic virus attacks the T4 cells of the immune system is this:
“If HIV is claimed to cause AIDS by killing T-cells, how is it possible that there is mass production of HIV in immortal T-cell lines as shown in the patent in 1984 as source of HIV proteins for “AIDS tests” by Gallo/NIH, Weiss/Burroughs Wellcome (UK), and Montagnier/Pasteur?”If the gallo-HIV actually attacks these infected cell lines, how come they are still producing HIV 21 years later! Here the proponents of the gallo-HIV theory have been saying excactly the opposite - that HIV does not kill cells, just like all other retroviruses. But that does not seem to bother those who hold the gallo-HIV dogma sacred, nor has it stirred a controversy or outcry in the mainstream media!
HIV is not behaving like a typical sexually transmitted disease
There is only one possible conclusion: as Japanese physician Y. Shiokawa has suggested, it is probable that drug use, multiple concurrent diseases, malnutrition, and other immunosuppressive factors are required to increase susceptibility. In fact, it better fits a model based on oxidative stress in cases of malnutrition and selenium deficiency and on oxidative damage and oxidative injury to cells of the immune system and cells in organs targeted by recreational drugs or immunotoxic medication.
The most difficult aspect of the HIV postulate is to have first decided that their HIV virus is an aggressive pathogen - which they claim targets the immune system itself, as HIV was said to infect the key CD4+ T cells that regulate the immune response, modifying or destroying their ability to function - and to reconcile this ’science’ with scientific data and evidence that some people “appear better able than others to resist progression of HIV infection or developing AIDS,” resulting in “long-term survivors” who can be divided into three groups;
1. Long-term nonprogressors who maintain healthy or steady levels of CD4+ T cells despite many years of infection,2. those tested-HIV-positive individuals who lose a significant proportion of CD4+ T cells but remain healthy, and
3. the people who remain uninfected despite repeated “exposure to HIV”.
So, to save the HIV postulate for AIDS they also claim that, once the virus infects CD4+ T cells, the virus’ genetic material is permanently integrated into the cell’s chromosomes, establishing permanent latency within infected cells. After infection, the HIV incorporates its genetic material into the host cell DNA. If a cell reproduces itself, each new cell also contains the integrated HIV genes. The virus can hide its genetic material for prolonged periods until the cell is activated and makes new viruses. So, its not an aggressive pathogen.
They also claim that other cells act as HIV reservoirs, harboring intact viruses that may remain undetected by the immune system while it “targets” the cells of the immune system.
There is no explaination on how an infected cell remains normal and remains undetected as an abnormal cell by NK cells or activated macrophages after the HIV incorporates its genetic material into the chromosomes of the cell. Such a virus, with such a capability, having a sophisticated enzyme system to incorporate its genetic material into the cells’ chromosomes and activate it later on into replicating itself cannot be so small and elusive that it avoids isolation and replication by other virologists. It must have a large amount of genetic material to do all of those things but retroviruses, such as the fictitious gallo-HIV, are “gifted” with too little genetic material.
The causal relationship between HIV and any disease is not settled
“HIV is an ordinary retrovirus. There is nothing about this virus that is unique. Everything that is discovered about HIV has an analogue in other retroviruses that don’t cause AIDS. HIV only contains a very small piece of genetic information. There is no way it can do all these elaborate things they say it does,” according to Dr. Harvey Bialy (Molecular Biologist and former editor of Bio/Technology and Nature Biotechnology as reported in Spin June 1992). Dr. Gordon Stewart (Emeritus Professor of Public Health, University of Glasgow) agrees that “AIDS is a behavioral disease. It is multifactorial, brought on by several simultaneous strains on the immune system - drugs, pharmaceutical and recreational, sexually transmitted diseases, multiple viral infections,” (Spin June 1992). Dr. Alfred Hässig, (1921-1999) was professor emeritus in immunology at the University of Bern, Director of the Swiss Red Cross Transfusion Service, and President of the Board of Trustees of the International Society of Blood Transfusion. His Swiss research group doesn’t believe that HIV causes AIDS either.
So, how did the world get duped into believing that HIV causes AIDS? Very simply but on a very serious note, one of the reasons is aptly stated by Dr. Richard Strohman (Emeritus Professor of Cell Biology at the University of California at Berkeley) when he said, “In the old days it was required that a scientist address the possibilities of proving his hypothesis wrong as well as right. Now there’s none of that in the standard HIV-AIDS program with all its billions of dollars,” (Penthouse April 1994).
Work by scientists, shoddy clinical trials and highly improper statistics concerning HIV and AIDS have been passed off as science. Unsuspectingly, non-governmental organizations, medical practitioners and the top members of the scientific establishment have carelessly supported or joined the media in spreading misinformation about the nature of AIDS and HIV as the pathogenic factor in AIDS although there are hundreds of thousands of healthy people who tested HIV-positive and live as long as HIV-negative people and there are many people with non-HIV AIDS who respond to proper nutrition.
And the ‘establishment’ has created bigger problems. There is no Universal Gold Standard ‘HIV’ test to prove ‘HIV’ positivity. The ‘HIV’ antibody test does not detect a ‘virus’ but an assortment of proteins that are non-specific to the hypothetical ‘HIV’. The proteins that are used in the ‘HIV’ test are merely the biological outcome of stressed white blood cells used in the lab. There can be no Gold Standard ‘HIV’ test because there is no Gold Standard ‘HIV’ isolate. Methods of classical virology require that all evidence of ‘HIV’ positivity must be confirmed by pure culturing of a patient’s lymphocytes and detection of whole, cell-free viral particles; so far this has never been achieved. The proteins that are used in the ‘HIV’ test are merely the biological outcome of stressed white blood cells used in the lab. In ‘Bio/Technology’, June 1993, ‘Aids’ analyst, Dr Eleni Papadopulos exposed the non-specificity and unreliability of the ‘HIV’ ‘antibody test’.
‘HIV’ is an artefact of cell-culture invented by Dr Robert Gallo. The phenomena collectively known as ‘HIV’ are non-specific: reverse transcriptase is non-specific; PCR is non-specific; Viral Load is non-specific. Each property relating to ‘HIV’ can be shown to pertain to the cells used in co-cultivation experiments. No particle of ‘HIV’ has ever been obtained pure, free of contaminants; nor has a complete piece of ‘HIV’ RNA (or the transcribed DNA) ever been proved to exist. Moreover, Dr David Ho admits that 99.8 per cent of putative ‘HIV particles’ are non-infectious; the remaining 0.2 per cent of ‘viral particles’, being defective, are not capable of replication. As a transmittable entity, ‘HIV’ could not survive in nature. This indicates that what we are calling ‘HIV’ is a misinterpreted, non-transmissible, endogenous epiphenomenon that should never have been classed as a virus.
Dr John Papadimitriou states that the proper controls have never been done: “They have not proven that they actually have detected a unique, exogenous retrovirus. The critical data to support that idea have not been presented. You have to be absolutely certain that what you have detected is unique and exogenous, and a single molecular species…(’Aids: The failure of contemporary science’, Neville Hodgkinson, Fourth Estate, 1996, page 375). Since 1989, detection of a 24,000 molecular weight protein (p24) in cell cultures, (T cells from persons presumed to be infected), or co- cultures, (of T cells from persons presumed to be infected, with T cells from normal individuals), has been used to quantify HIV in cells, “cellular viremia” (Masquelier et al., 1992). Detection of p24 in cultures of T cells from normal individuals with plasma from those presumed to be infected has been used to quantify HIV in plasma, “plasma viremia” (Coombs et al., 1989; Ho et al., 1989; Clark et al., 1991). There are many reasons why p24 cannot be used to quantify or even detect the presence of “HIV infectious particles”. There is ample evidence that the p24 protein is not HIV specific (Papadopulos-Eleopulos et al., 1993a).
Look at some of the key points, as follows:
1. HIV is not behaving like a typical sexually transmitted disease.2. Other immunosuppressive factors are required to increase susceptibility.
3. “AIDS is a behavioral disease. It is multifactorial, brought on by several simultaneous strains on the immune system - drugs, pharmaceutical and recreational, sexually transmitted diseases, multiple viral infections.”
4. There is nothing about this virus that is unique. Everything that is discovered about HIV has an analogue in other retroviruses that don’t cause AIDS. HIV only contains a very small piece of genetic information.
5. Like in all other viral infections, eg smallpox, you have to be absolutely certain that what you have detected is unique and exogenous, and a single molecular species, not particles.
6. The ‘HIV’ antibody test does not detect a ‘virus’ but an assortment of proteins that are non-specific to the hypothetical ‘HIV’. The proteins that are used in the ‘HIV’ test are merely the biological outcome of stressed white blood cells used in the lab.
7. “99.8 per cent of putative ‘HIV particles’ are non-infectious; the remaining 0.2 per cent of ‘viral particles’, being defective, are not capable of replication. As a transmittable entity, ‘HIV’ could not survive in nature”.
8. “It is multifactorial, brought on by several simultaneous strains on the immune system - drugs, pharmaceutical and recreational, sexually transmitted diseases, multiple viral infections.”
9. Gallo claimed that the interaction of gp41 with antibodies found in AIDS patient sera is proof that gp41 is coded by the “HIV genome”, and that both gp41 and the antibodies are specific to a retrovirus.
10. The Epstein-Barr virus (EBV) resides as a persistent infection in human leukocyte antigen (HLA) class II+ B lymphocytes and is associated with a number of malignancies. The EBV lytic-phase protein gp42 serves at least two functions: gp42 acts as the coreceptor for viral entry into B cells and hampers T-cell recognition via HLA class II molecules through steric hindrance of T-cell receptor-class II-peptide interactions (Maaike E. Ressing et al, Epstein-Barr Virus gp42 Is Posttranslationally Modified To Produce Soluble gp42 That Mediates HLA Class II Immune Evasion, Journal of Virology, January 2005, p. 841-852, Vol. 79, No. 2).
11. In 1980, two research groups, one from the Laboratory of Cellular and Molecular Biology, National Cancer Institute and the other from the Laboratory of Viral Oncology, Memorial Sloan-Kettering Cancer Center, using the “viral glycoproteins”, found that the antibodies present in human sera which reacted with these proteins were “directed against carbohydrate structures” and concluded that “The results are consistent with the idea that the antibodies in question are elicited as a result of exposure to many natural substances possessing widely cross-reacting antigens and are not a result of widespread infection of man with replication competent oncoviruses”.
12. Exposure of RAW 264.7 macrophages to JP, a cell permeant analog of phalloidin that increases and stabilizes polymerized actin in living cells, reduced the ability of RAW 264.7 macrophages to phagocytose fluorescent Klebsiella by 50%. This indicates that increased actin polymerization is a potential mechanism explaining impairment of phagocytosis by oxidative stress and since AIDS is a condition caused by excess free radicals (in malnourished people) (see Philip J et al, Hyperoxia Impairs Antibacterial Function of Macrophages Through Effects on Actin, American Journal of Respiratory Cell and Molecular Biology. Vol. 28, pp. 443-450, 2003), it quite clearly proves that oxidative stress on macrophages leads to increased actin polymerisation and formation of prominent stress fibers and actin aggregates which could occur in people recovering from malaria, influenza or in people suffering from chronic fatigue due to mitochodrial oxidative stress or ethanol toxicity or drug induced oxidative stress.
13. The serological diagnosis of HIV infection is usually made on the basis of the detection of circulating antibodies specific for viral antigens gp41, gp120 and gp160 and possibly gp42. Studies show them to be non-viral glycoproteins or polymer actins that have suffered cleavage at different points. Quite possibly hydrogen peroxide that accumulates in people who are low in glutathione or the seleno-antioxidant enzyme that converts it into water and oxygen, can cause cleavage in polymer actins produced by cells of the immune system under oxidative stress. With relatively lower oxygen generated by the enzymatic conversion of hydrogen peroxide, people with chronic malnutrition and low selenium intake could create a cellular environment of hyperoxia thereby initiating marked changes, including an increase in the degree of actin polymerization, loss of cortical actin, and the formation of prominent stress fibers and actin aggregates.
What appears to be consistent is the observation supported by scientific tests that the viral antigens in the Gallo Isolate are actin polymers produced by cells or white blood cells in oxidative stress and consistent with the free radical theory of AIDS that oxidative stress produces a broad range of symptoms and illnesses and that includes suppression of the immune system or immunodeficiency. One certainty that emerges is that this diametrically opposes the claim of a specific disease caused by a specific virus that has antigenic specificity like other viruses.
But could the body be producing antibodies to these polymer actins produced under oxidative stress and hence being recognized as non-self or could some of these actins especially gp41 and gp42 act as conjugating glycoproteins that bind with EBV viral genomes that can hide in cells of the immune system and that conjugate is later broken by excess hydrogen peroxide during chronic oxidative stress or severe oxidative stress caused by chemicals and drugs? Quite possibly both, which means that AIDS is more likely to be an EBV latency disease triggered by oxidative stress, whether by chronic malnutrition or drugs. That does explain the different rates of “progression” of AIDS and opportunistic infections in the very poor societies and in the developed world (see: THE EPSTEIN-BARR VIRUS IN AIDS on www.newmediaexplorer.org/sepp - scroll down three pages ).
On one hand their HIV-causes-AIDS hypothesis tells people that after the HIV infects cells in the immune system, it replicates in them and it leads to the ultimate devastation of the immune system while on the other the prescription is primarily large doses of immunotoxic and immunosuppresive “medicine”!
WHY is this enigma approved by the medical authorities? Other drugs such as, sulfonamides and trimethoprim are also used in the treatment of PCP, which cause severe hematological complications, including agranulocytosis, hemolytic and megaloblastic anemia and thrombocytopenia. The results of clinical trials on AZT and protease inhibitors have revealed that these agents are poisons and not cures. AZT is a very toxic poison that promotes cancer formation in the human body. “Granulocytopenia”, is a deficiency of the most numerous cells of our immune system, which in turn leads to opportunistic infections. Prolonged use of AZT attacks the immune system through free radical toxicity.
And now, the situation as described by Dr. Roger Cunningham (Immunologist, Microbiologist and Director of the Centre for Immunology at the State University of New York at Buffalo) has become rather grave because “Unfortunately, an AIDS ‘establishment’ seems to have formed that intends to discourage challenges to the dogma on one side and often insists on following discredited ideas on the other,” (Sunday Times (London) 3 April 1994).
The greatest intrigue remains locked in the question - “How is it possible for the mass production of HIV in immortal T-cell lines to continue, if Gallo claims that they attack these cells as pathogens?” Then comes another riddle - “How is a small retrovirus, with so little genetic material, able to infect and incorporate itself into the cell DNA and later become virulent, killing it?”
The gallo-HIV enters the Guiness Book Of Records as the tiniest fictitious artefact that ever created a multi-billion dollar industry.
Ooops, I forgot, we are not supposed to question the gallo-HIV dogma. Science is meant to be applied elsewhere, not within the haloed confines of the gallo-HIV disease and the AIDS industry it spurned.
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Some other articles of Beldeu Singh that can be found on this site:
AIDS, NON-HIV AIDS AND PRESCRIPTION AIDS
WHY OMEGA-3 FISH OIL PROTECTS YOUR HEART AND BRAIN
Flowers and Spices: Ayurveda or Toxic Chemotherapy?
- - -WORLD AIDS DAY MESSAGE FROM DR. LEO REBELLO
1st December is a World AIDS Day -- AIDS Racket Day would be an apt description. My 10-point advice to you is given below :
1.. HIV has NOT been identified. Therefore to say that it causes AIDS is the biggest con of our time.
2.. AIDS is NOT a new disease. It is called Oja-kshaya in Ayurveda, Vettai Noi in Siddha and Al-Zabool in Unani. It is due to depleted immunity.
3.. AIDS is NOT (repeat not) caused by normal/natural sex. So enjoy your natural sex without guilt or stress. Because by creating scare they create psycho-neuro immunologic defects in your system.
4.. The only known cause of AIDS is antibiotics, anti-retroviral drugs, vaccines and other deadly carcinogenic chemicals that are indiscriminately being given to all and sundry. It is madness to take medicines for anything and everything. Medicines do not heal, they kill.
5.. I am NOT against condoms per se. But please note that condoms offer only 60% protection. But because of over exposure to condoms, today, boys and girls as young as 12 are experimenting with sex, leading to condom culture, condom civilisation, condom ethics, condom morals and condemned society.
6.. Remember that all diseases start and end with Diet and Stress. So take care what you eat, and what you think and how you work or react.
7.. Beware of Vaccines. They are the root cause of Tuberculosis, Cancers, Autism AIDS and other debilitating diseases. If at all, you may take Homeopathic Nosodes, Urine Vaccine, Veggie Vaccines or Auto-Blood Vaccines.
8.. For Venereal Diseases use Herbal pastes, pessaries and douches and maintain physical and mental hygiene.
9.. Eat well, sleep well, meditate, play music, dance, exercise, laugh, take part in sports, go for movies to relax and re-create yourself.
10. Dr. Water, Dr. Diet, Dr. Sleep, Dr. Exercise, Dr. Sunshine, Dr. Humour and Dr. Leo Rebello are the seven most wonderful doctors of the world. If you follow their advice properly, you will live to be healthy hundred without ills, pills and doctors bills.
LET US WORK FROM AIDS SCARE TO AIDS CARE.
For more details read my books, more particularly, AIDS AND ALTERNATIVE
MEDICINESee also:
Selenium pills 'may combat HIV'
The University of Miami found a lower HIV viral load in patients who took selenium supplements for nine months. Selenium deficiencies have been recorded in HIV patients, and evidence suggests the mineral can improve the function of the immune system.World Aids Day - Do the facts matter?
[The following from Sharon Stone to Larry King]
"a child is dying every two minutes from AIDS. We have to look at what's really happening. And I think the biggest number we have to look at is how many people have survived AIDS. Zero."
…
"people don't really believe it can happen to them. I don't think that people are in the reality of how prevalent AIDS really is and how serious that it really is. . . . it's the fourth leading killer of women in America . . . half of the people that have AIDS are women."
…
Medications prevent mother-to-child transmission: "mothers of HIV [sic] were able to give birth to zero children with AIDS".Henry Bauer, who wrote a book: The Origin, Persistence and Failings of HIV/AIDS Theory discusses the actual numbers around HIV and Aids in an intelligent, non-emotional manner. Perhaps it is time we paid attention to people like him.
posted by Sepp Hasslberger on Thursday November 30 2006
updated on Saturday December 4 2010URL of this article:
http://www.newmediaexplorer.org/sepp/2006/11/30/world_aids_day_time_to_rethink_prevention_treatment.htm
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August 29, 2005 - Sepp Hasslberger
